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<script>on mouseUpvisual effect wipe right slowgo previous cdend mouseUp</script>
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<script>on mouseUpanswer "There is no vaccine for A.I.D.S. yet."end mouseUp</script>
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<content>
<layer>card</layer>
<id>1</id>
<text>With the Helper T-cell lymphocyte ruptured, it collapses. A flood of new viral cells enter the blood stream and invade more tissues and other Helper T-cells. The supply of Helper T-cells becomes depleted, and the abnormally high concentration of Supressor cells restricts B-cell antibody production. The compromised immune system is unable to resist further invasion by other pathogens. As the disease progresses its victims suffer the effects of a failing immune system. The HIV virus continues to spread, resistance to opportunistic diseases decreases, and the patient suffers from increasingly severe symptoms.</text>
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<name></name>
<script>on idlerepeat with i= 0 to 9send idle to cd btn ("HIV" &i)end repeatpass idleend idle</script>
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card_7329.xml
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<script>on mouseUpvisual effect wipe left slowgo next cdend mouseUp</script>
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<script>on mouseUpvisual iris open to whitevisual dissolve to blackgo cd 1end mouseUp</script>
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<text>The cell produces the virus until all the virus dies or all host cell material is used up. Once reactivated, progeny virons leave the cell by budding. Because Helper T-cell cytoplasm is depleted in the process, the cell dies.</text>
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<text><span class="style1">The reverse trans- criptase tricks the host cell's </span><span class="style2">DNA </span><span class="style1">into producing viral replication messages, thus reverse trans- criptase makes a DNA copy of the viral RNA. The copy DNA enters the T-cell nucleus. The viral genes take over the genes of the infected cell. Then for a variable period of time, the virus is latent, during which time it may be passed on to the daughter cells during cell division.</span></text>
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<text>Shortly after the viral enzymes have entered the cell, the viral nucleic acid (RNA) begins to replicate itself and synthesize the virus proteins using these enzymes and the raw materials of the cell. Instead of causing change or abnormal growth of the Helper T-cells, HIV uses up the host cells while replicating.</text>
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<text><span class="style1">he HIV viral envelope is covered with spikes of glycoprotein. After entering the host, a viral particle uses at least one of these spikes ( </span><span class="style2">GP120</span><span class="style1"> ) to attach itself to a receptor (</span><span class="style2">CD4</span><span class="style1">) on the Helper T-cell lymphocyte molecule. The process of infection begins.</span></text>
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<text><span class="style1">esearchers have discovered that HIV infects far faster than previously sus-pected, perhaps as quickly as within 4 hours of viral contact! Infection may occur after sexual contact with an in-fected person (even via artifical in-semination) or exposure through dis-eased or broken skin to contaminated blood or body fluids. Since </span><span class="style2">HIV</span><span class="style1"> primarily infects the Helper T-cell lymphocytes, lymphocytic tissues such as blood, semen, and vaginal secretions probably contain higher concen- trations of the virus and are more infective. Whether or not an infected individual develops the disease, he is a carrier of the virus, and AIDS carriers are infectious to others. Some evidence suggests that asymptomatic HIV carriers may have higher viral titers than those who have already contracted the disease.</span></text>
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<text><span class="style1">ell mediated immunity (T cell immunity) is the body's usual response to fungus, viral, or mycobacterial infections. This type of immune response is mediated by small lymphocytes and is dependent on the presence of the thymus at birth. T cells must pass through the thymus gland before they are deposited in lymphoid tissue. When T cells leave the thymus they circulate in the blood as long-lived small and medium sized lymphocytes with a life span up to 5 years. </span><span class="style2">Helper T-Cells</span><span class="style1"> stimulate the production of B-cells which produce antibodies to fight an invader. When the pathogen invader is destroyed, Supressor T-cells stop B-cell production to restore equi- librium. In a healthy individual the Helper/Supressor T-cell ratio is 2:1. In the AIDS victim the ratio may be reversed.</span></text>
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<text>Theoretically any cell with a CD4 receptor on its cell membrane can be infected by HIV. Biopsy specimens have shown that most cells composing the colorectal tissues can be directly infected. The tiny glial cells of the nervous system appear to be susceptible. The brain's macrophages may also be infected by HIV. Connective tissue fibroblasts have been infected in the laboratory leading to speculation that these cells can become reservoirs of infection. Marrow cells and immature monocytes have also been infected in the lab suggesting a mechanism for bypassing the immune system and releasing large numbers of virions at one time. However, lymphocytes remain the most easily infected cells. </text>
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<text>This is the cytoplasm of the lymphocyte cell, the protoplasm minus the nucleus. It is unique in that it possesses a certain group of properties with which no combination of non-living substances is endowed. Here in human cells other structures (not shown) such as ribosomes, lysosomes, centrioles, Golgi apparatus, endoplasmic reticulum, and mitochondrion can be found. When infected by a virus, the cell's own energy sources, its ribosomes and other raw materials, will be used to synthesize viral proteins.</text>
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<text>This central, usually oval mass of protoplasm present in human cells (and most plant and animal cells) contains most of the hereditary material and is necessary for such functions as growth and reproduction. Here the rod shaped bodies known as chromosomes, made of highly coiled and condensed DNA threads, protein, and small amounts of RNA are found. After infection, the reverse transcriptase of the Human Immundeficiency Virus tricks the host cell's DNA into producing viral replication messages.</text>
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<text><span class="style1">he </span><span class="style2">HIV RETROVIRUSES</span><span class="style1">have a dense nucleus containing core proteins</span><span class="style2"> P</span><span class="style1">, reverse transcriptase </span><span class="style2">RT</span><span class="style1">, and </span><span class="style2">RNA</span><span class="style1"> . One of the core proteins, </span><span class="style2">P1</span><span class="style1">, lies within the lipid envelope that surrounds the nucleus and forms the viron matrix. The envelope is slightly more than 100 nanometers in diameter. Spikes of envelope glycoprotein ( </span><span class="style2">GP12</span><span class="style1"> & </span><span class="style2">GP4</span><span class="style1"> ) are found on the cell's surface. </span><span class="style2">GP12</span><span class="style1"> attaches itself to a host cell's </span><span class="style2">CD</span><span class="style1"> receptor to begin the process of infection. Like all retroviruses, HIV has no DNA of its own and replicates by releasing its viral enzyme, reverse transcriptase, into the cell it has invaded.</span></text>
